Physiologic Biomarkers to Detect Subclinical Acute Kidney Injury in Premature Infants
Acute kidney Injury (AKI) prior to the completion of nephrogenesis at 34 weeks’ gestation has significant life-long effects. The immature kidney only receives 3-4% of total cardiac output, compared to 20% in term infants, children and adults. Therefore, minimal decreases in oxygen delivery may substantially compromise proper oxygen utilization increasing the risk for morphologic changes and reduced nephron endowment. Current diagnostic criteria (serum creatinine (sCr) elevations with oliguria) cannot detect early-onset AKI, as up to 50% of nephron damage has already occurred by the time these abnormalities become apparent. This presentation will look at new research related to the current diagnostic criteria for AKI in the preterm infant, the physiologic mechanisms involved in AKI and short and long-term implications.
This presentation was originally offered at our GOLD Neonatal Online Conference 2022.
Dr. Marin is currently an Assistant Professor at Augusta University, and is an active researcher in the Level IV NICU at Children’s Hospital of Georgia. She received her BSN from the University of Tennessee, her MSN from Stony Brook and her PhD from Emory University. Dr. Marin’s program of research is focused on defining non-invasive methods to predict early-onset acute kidney injury in preterm infants, including analysis of metabolomics, proteomics, the gut-kidney microbiome axis, and renal hypoxia measured by near-infrared spectroscopy as they relate to subclinical and actual acute kidney disease.
1. Describe current diagnostic criteria for Acute Kidney Injury (AKI) in the preterm and term infant.
2. Identify the physiologic mechanisms involved in renal oxygenation measurement using near-infrared spectroscopy technology.
3. Describe the short and long-term implications associated with AKI development in the preterm population.
Acute kidney Injury (AKI) prior to the completion of nephrogenesis at 34 weeks’ gestation has significant life-long effects. The immature kidney only receives 3-4% of total cardiac output, compared to 20% in term infants, children and adults. Therefore, minimal decreases in oxygen delivery may substantially compromise proper oxygen utilization increasing the risk for morphologic changes and reduced nephron endowment. Current diagnostic criteria (serum creatinine (sCr) elevations with oliguria) cannot detect early-onset AKI, as up to 50% of nephron damage has already occurred by the time these abnormalities become apparent. This presentation will look at new research related to the current diagnostic criteria for AKI in the preterm infant, the physiologic mechanisms involved in AKI and short and long-term implications.
Accreditation
CERPs - Continuing Education Recognition Points
Applicable to IBCLC Lactation Consultants, Certified Lactation Consultants (CLCs), CBEs, CLE, Doulas & Birth Educators. GOLD Conferences has been designated as a Long Term Provider of CERPs by IBLCE--Approval #CLT114-07. This program is approved for 1 R-CERP.
Nurse Contact Hours
This nursing continuing professional development activity was approved by the American Nurses Association Massachusetts, an accredited approver by the American Nurses Credentialing Center’s Commission on Accreditation for 1 Nursing Contact Hour.
Nurse Contact Hours are valid until 05/31/2024.
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Additional Details
Viewing Access Time: 2 Weeks
Tags / Categories
(IBCLC) Development and Nutrition, (IBCLC) Infant, (IBCLC) Infant, (IBCLC) Pathology, Neonatal Illness, Neonatology
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